Regression of Atherosclerosis and Smooth Muscle Cell Proliferation
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چکیده
منابع مشابه
The effect of adrenomedullin and proadrenomedullin N- terminal 20 peptide on angiotensin II induced vascular smooth muscle cell proliferation
Objective(s): The study aimed to investigate the effects of adrenomedullin (ADM) and proadrenomedullin N- terminal 20 peptide (PAMP) on angiotensin II (AngII)-stimulated proliferation in vascular smooth muscle cells (VSMCs). Materials and Methods: Thoracic aorta was obtained from Wistar rats and VSMCs were isolated from aorta tissues and then cultured. In vitro cultured VSMCs were stimulated w...
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1. Bentham J, Rodriguez-Arnao J, Ross RJM. Acquired growth hormone resistance in patients with hypercatabolism. Harm Res. 1993;40:87-91. 2. Ross RJM, Chew SL. Acquired growth hormone resistance. Eur J Endocrinol. 1995;132:655-660. 3. Anker SD, Chua TP, Swan JW, Ponikowski P, Harrington D, Kox WJ, Poole-Wilson PA, Coats AJS. Hormonal changes and catabolic/anabolic imbalance in chronic heart fail...
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1. Bentham J, Rodriguez-Arnao J, Ross RJM. Acquired growth hormone resistance in patients with hypercatabolism. Harm Res. 1993;40:87-91. 2. Ross RJM, Chew SL. Acquired growth hormone resistance. Eur J Endocrinol. 1995;132:655-660. 3. Anker SD, Chua TP, Swan JW, Ponikowski P, Harrington D, Kox WJ, Poole-Wilson PA, Coats AJS. Hormonal changes and catabolic/anabolic imbalance in chronic heart fail...
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Markers of cell senescence have been identified in both the blood and vessel wall of patients with atherosclerosis. In particular, vascular smooth muscle cells (VSMCs) derived from human plaques show numerous features of senescence both in culture and in vivo. This review summarises the evidence for VSMC senescence in atherosclerosis, and outlines the mechanisms and triggers leading to their se...
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Vascular smooth muscle cells (VSMCs) exhibit phenotypic and functional plasticity in order to respond to vascular injury. In case of the vessel damage, VSMCs are able to switch from the quiescent 'contractile' phenotype to the 'proinflammatory' phenotype. This change is accompanied by decrease in expression of smooth muscle (SM)-specific markers responsible for SM contraction and production of ...
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ژورنال
عنوان ژورنال: The Journal of Japan Atherosclerosis Society
سال: 1991
ISSN: 0386-2682,2185-8284
DOI: 10.5551/jat1973.19.8_743